HLA-G-mediated Immune Tolerance: Past and New Outlooks

The non-classical HLA class I molecule HLA-G is different from classical HLA class I molecules because of the low polymorphism in the coding region, the fact that HLA-G primary transcript is alternatively spliced in seven isoforms, and the inhibitory action on immune cells. Although HLA-G is low pol...

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Główni autorzy: Silvia Gregori, Joel LeMaoult
Format: Online
Język:angielski
Wydane: Frontiers Media SA 2021
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Dostęp online:22970
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author Silvia Gregori
Joel LeMaoult
author_browse Joel LeMaoult
Silvia Gregori
author_facet Silvia Gregori
Joel LeMaoult
author_sort Silvia Gregori
collection Directory of Open Access Books
description The non-classical HLA class I molecule HLA-G is different from classical HLA class I molecules because of the low polymorphism in the coding region, the fact that HLA-G primary transcript is alternatively spliced in seven isoforms, and the inhibitory action on immune cells. Although HLA-G is low polymorphic, variants in both promoter and 3’ un-translated region (UTR) of HLA-G locus regulate its expression. In healthy conditions, a basal level of HLA-G gene transcription is observed in most cells and tissues; however, translation into HLA-G protein is restricted to trophoblasts in the placenta, where it participates in promoting tolerance at the fetal-maternal interface. HLA-G is also expressed by thymic epitelial, cornea, mesenchymal stem cells, nail matrix, pancreatic beta cells, erythroid, and endothelial precursors. HLA-G can be neo-expressed in adult tissues in pathological conditions, and its expression has been documented autoimmune disorders, viral infections, and cancer. In the latter setting de novo HLA-G expression is associated with the capability of tumor cells to evade the immune control. In the last decade it has become evident that HLA-G expression on T cells and antigenpresenting cells confers to these cells tolerogenic properties. This Research Topic focused on i) summarizing updated clinical and immunological evidences that HLA-G expression is associate with beneficial or detrimental tolerance, ii) gathering new insights into the mechanisms governing the expression of HLA-G in healthy and pathological conditions, such as pre-eclampsia, and iii) examining the mechanisms underlying HLA-G mediated tolerance.
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spelling doab-20.500.12854ir-494872024-03-30T23:21:42Z HLA-G-mediated Immune Tolerance: Past and New Outlooks Silvia Gregori Joel LeMaoult R5-920 RC581-607 Pregnancy Autoimmunity Immuno-modulation Pre-Eclampsia Infections Exosomes HLA-G polymorphisms tolerance Cancer thema EDItEUR::M Medicine and Nursing The non-classical HLA class I molecule HLA-G is different from classical HLA class I molecules because of the low polymorphism in the coding region, the fact that HLA-G primary transcript is alternatively spliced in seven isoforms, and the inhibitory action on immune cells. Although HLA-G is low polymorphic, variants in both promoter and 3’ un-translated region (UTR) of HLA-G locus regulate its expression. In healthy conditions, a basal level of HLA-G gene transcription is observed in most cells and tissues; however, translation into HLA-G protein is restricted to trophoblasts in the placenta, where it participates in promoting tolerance at the fetal-maternal interface. HLA-G is also expressed by thymic epitelial, cornea, mesenchymal stem cells, nail matrix, pancreatic beta cells, erythroid, and endothelial precursors. HLA-G can be neo-expressed in adult tissues in pathological conditions, and its expression has been documented autoimmune disorders, viral infections, and cancer. In the latter setting de novo HLA-G expression is associated with the capability of tumor cells to evade the immune control. In the last decade it has become evident that HLA-G expression on T cells and antigenpresenting cells confers to these cells tolerogenic properties. This Research Topic focused on i) summarizing updated clinical and immunological evidences that HLA-G expression is associate with beneficial or detrimental tolerance, ii) gathering new insights into the mechanisms governing the expression of HLA-G in healthy and pathological conditions, such as pre-eclampsia, and iii) examining the mechanisms underlying HLA-G mediated tolerance. 2021-02-11T15:28:14Z 2021-02-11T15:28:14Z 2017-07-06 13:27:36 2017 book 22970 16648714 9782889451197 https://directory.doabooks.org/handle/20.500.12854/49487 eng Frontiers Research Topics image/jpeg Attribution 4.0 International http://www.frontiersin.org/books/HLA-G-mediated_Immune_Tolerance_Past_and_New_Outlooks/1125#nogo http://journal.frontiersin.org/researchtopic/2452/hla-g-mediated-immune-tolerance-past-and-new-outlooks Frontiers Media SA 10.3389/978-2-88945-119-7 10.3389/978-2-88945-119-7 bf5ce210-e72e-4860-ba9b-c305640ff3ae 9782889451197 92 open access
spellingShingle R5-920
RC581-607
Pregnancy
Autoimmunity
Immuno-modulation
Pre-Eclampsia
Infections
Exosomes
HLA-G
polymorphisms
tolerance
Cancer
thema EDItEUR::M Medicine and Nursing
Silvia Gregori
Joel LeMaoult
HLA-G-mediated Immune Tolerance: Past and New Outlooks
title HLA-G-mediated Immune Tolerance: Past and New Outlooks
title_full HLA-G-mediated Immune Tolerance: Past and New Outlooks
title_fullStr HLA-G-mediated Immune Tolerance: Past and New Outlooks
title_full_unstemmed HLA-G-mediated Immune Tolerance: Past and New Outlooks
title_short HLA-G-mediated Immune Tolerance: Past and New Outlooks
title_sort hla g mediated immune tolerance past and new outlooks
topic R5-920
RC581-607
Pregnancy
Autoimmunity
Immuno-modulation
Pre-Eclampsia
Infections
Exosomes
HLA-G
polymorphisms
tolerance
Cancer
thema EDItEUR::M Medicine and Nursing
topic_facet R5-920
RC581-607
Pregnancy
Autoimmunity
Immuno-modulation
Pre-Eclampsia
Infections
Exosomes
HLA-G
polymorphisms
tolerance
Cancer
thema EDItEUR::M Medicine and Nursing
url 22970
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