Lipopolysaccharides (LPSs)

The cytoplasm of Gram-negative bacteria is bound by three layers: an inner membrane, a layer of peptidoglycan, and an outer membrane. The outer membrane is an asymmetric lipidic bilayer, with phospholipids on its inner surface and lipopolysaccharides (LPSs) on the outside, with the latter being the...

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Հիմնական հեղինակ: Tomás, Juan M.
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Հրապարակվել է: MDPI - Multidisciplinary Digital Publishing Institute 2021
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Առցանց հասանելիություն:44738
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author Tomás, Juan M.
author_browse Tomás, Juan M.
author_facet Tomás, Juan M.
author_sort Tomás, Juan M.
collection Directory of Open Access Books
description The cytoplasm of Gram-negative bacteria is bound by three layers: an inner membrane, a layer of peptidoglycan, and an outer membrane. The outer membrane is an asymmetric lipidic bilayer, with phospholipids on its inner surface and lipopolysaccharides (LPSs) on the outside, with the latter being the major component of the outer leaflet and covering nearly three-quarters of the total outer cell surface. All LPSs possess the same general chemical architecture independently of bacterial activity (pathogenic, symbiotic, commensal), ecological niche (human, animal, soil, plant, water), or growth conditions. Endotoxins are large amphiphilic molecules consisting of a hydrophilic polysaccharide component and a covalently bound hydrophobic and highly conserved lipid component, termed lipid A (the endotoxin subunit). The polysaccharide component can be divided into two subdomains: the internal and conserved core region as well as the more external and highly variable O-specific chain, also referred to as the O-antigen due to its immunogenic properties. LPSs are endotoxins, one of the most potent class of activators of the mammalian immune system; they can be released from cell surfaces of bacteria during multiplication, lysis, and death. LPS can act through its biological center (lipid A component) on various cell types, of which macrophages and monocytes are the most important.
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spelling doab-20.500.12854ir-517782024-03-30T23:21:36Z Lipopolysaccharides (LPSs) Tomás, Juan M. R5-920 RC581-607 LAL and LER cornea Aeromonas tear fluid protein dynamics O-antigen Plesiomonas shigelloides lipopolysaccharide induced TNF? factor hepcidin pJNK rat Proteus penneri lipid A LPS tolerance iron regulatory protein 1 (IRP1) MALDI-TOF MS outer core oligosaccharide inhibitor design phagosome lipopolysaccharide NMR spectroscopy Bordetellae respiratory syncytial virus dendritic cell MAT GT-B polysorbate Edwardsiella tarda LPS innate immune d-galactan I inflammation aspirin lipopolysaccharide (LPS) collagen heptosyltransferase rhinovirus serum resistance hypothalamic inflammation NMR IL-6/JAK2/STAT3 pathway glycosyltransferase toll-like receptor core oligosaccharide Coxiella burnetii virenose oxidative stress ESI FT-ICR endotoxin Salmonella P65 (nuclear factor-?B) nitric oxide (NO) allergic respiratory disorder bacteria adhesion molecule ESI MSn endotoxaemia HR-MAS double-stranded RNA epithelial cell mouse lipopolysaccharide stimulation complement inner core oligosaccharide LPS aggregates Q fever O-acetylation sepsis chemokine hygiene hypothesis anti-conjugate serum structural determination Small Angle X-ray Scattering genomics mass spectrometry keratocyte insulin resistance Low Endotoxin Recovery phase transitions Megalobrama amblycephala Bordetella holmesii Erwinia amylovora structure time response fibroblast genomic sialic acid ESI MS reptile-associated salmonellosis thema EDItEUR::M Medicine and Nursing The cytoplasm of Gram-negative bacteria is bound by three layers: an inner membrane, a layer of peptidoglycan, and an outer membrane. The outer membrane is an asymmetric lipidic bilayer, with phospholipids on its inner surface and lipopolysaccharides (LPSs) on the outside, with the latter being the major component of the outer leaflet and covering nearly three-quarters of the total outer cell surface. All LPSs possess the same general chemical architecture independently of bacterial activity (pathogenic, symbiotic, commensal), ecological niche (human, animal, soil, plant, water), or growth conditions. Endotoxins are large amphiphilic molecules consisting of a hydrophilic polysaccharide component and a covalently bound hydrophobic and highly conserved lipid component, termed lipid A (the endotoxin subunit). The polysaccharide component can be divided into two subdomains: the internal and conserved core region as well as the more external and highly variable O-specific chain, also referred to as the O-antigen due to its immunogenic properties. LPSs are endotoxins, one of the most potent class of activators of the mammalian immune system; they can be released from cell surfaces of bacteria during multiplication, lysis, and death. LPS can act through its biological center (lipid A component) on various cell types, of which macrophages and monocytes are the most important. 2021-02-11T17:49:13Z 2021-02-11T17:49:13Z 2020-04-07 23:07:08 2020 book 44738 9783039282562 9783039282579 https://directory.doabooks.org/handle/20.500.12854/51778 eng application/octet-stream Attribution-NonCommercial-NoDerivatives 4.0 International https://mdpi.com/books/pdfview/book/2012 MDPI - Multidisciplinary Digital Publishing Institute 10.3390/books978-3-03928-257-9 10.3390/books978-3-03928-257-9 46cabcaa-dd94-4bfe-87b4-55023c1b36d0 9783039282562 9783039282579 390 open access
spellingShingle R5-920
RC581-607
LAL and LER
cornea
Aeromonas
tear fluid
protein dynamics
O-antigen
Plesiomonas shigelloides
lipopolysaccharide induced TNF? factor
hepcidin
pJNK
rat
Proteus penneri
lipid A
LPS tolerance
iron regulatory protein 1 (IRP1)
MALDI-TOF MS
outer core oligosaccharide
inhibitor design
phagosome
lipopolysaccharide
NMR spectroscopy
Bordetellae
respiratory syncytial virus
dendritic cell
MAT
GT-B
polysorbate
Edwardsiella tarda
LPS
innate immune
d-galactan I
inflammation
aspirin
lipopolysaccharide (LPS)
collagen
heptosyltransferase
rhinovirus
serum resistance
hypothalamic inflammation
NMR
IL-6/JAK2/STAT3 pathway
glycosyltransferase
toll-like receptor
core oligosaccharide
Coxiella burnetii
virenose
oxidative stress
ESI FT-ICR
endotoxin
Salmonella
P65 (nuclear factor-?B)
nitric oxide (NO)
allergic respiratory disorder
bacteria
adhesion molecule
ESI MSn
endotoxaemia
HR-MAS
double-stranded RNA
epithelial cell
mouse
lipopolysaccharide stimulation
complement
inner core oligosaccharide
LPS aggregates
Q fever
O-acetylation
sepsis
chemokine
hygiene hypothesis
anti-conjugate serum
structural determination
Small Angle X-ray Scattering
genomics
mass spectrometry
keratocyte
insulin resistance
Low Endotoxin Recovery
phase transitions
Megalobrama amblycephala
Bordetella holmesii
Erwinia amylovora
structure
time response
fibroblast
genomic
sialic acid
ESI MS
reptile-associated salmonellosis
thema EDItEUR::M Medicine and Nursing
Tomás, Juan M.
Lipopolysaccharides (LPSs)
title Lipopolysaccharides (LPSs)
title_full Lipopolysaccharides (LPSs)
title_fullStr Lipopolysaccharides (LPSs)
title_full_unstemmed Lipopolysaccharides (LPSs)
title_short Lipopolysaccharides (LPSs)
title_sort lipopolysaccharides lpss
topic R5-920
RC581-607
LAL and LER
cornea
Aeromonas
tear fluid
protein dynamics
O-antigen
Plesiomonas shigelloides
lipopolysaccharide induced TNF? factor
hepcidin
pJNK
rat
Proteus penneri
lipid A
LPS tolerance
iron regulatory protein 1 (IRP1)
MALDI-TOF MS
outer core oligosaccharide
inhibitor design
phagosome
lipopolysaccharide
NMR spectroscopy
Bordetellae
respiratory syncytial virus
dendritic cell
MAT
GT-B
polysorbate
Edwardsiella tarda
LPS
innate immune
d-galactan I
inflammation
aspirin
lipopolysaccharide (LPS)
collagen
heptosyltransferase
rhinovirus
serum resistance
hypothalamic inflammation
NMR
IL-6/JAK2/STAT3 pathway
glycosyltransferase
toll-like receptor
core oligosaccharide
Coxiella burnetii
virenose
oxidative stress
ESI FT-ICR
endotoxin
Salmonella
P65 (nuclear factor-?B)
nitric oxide (NO)
allergic respiratory disorder
bacteria
adhesion molecule
ESI MSn
endotoxaemia
HR-MAS
double-stranded RNA
epithelial cell
mouse
lipopolysaccharide stimulation
complement
inner core oligosaccharide
LPS aggregates
Q fever
O-acetylation
sepsis
chemokine
hygiene hypothesis
anti-conjugate serum
structural determination
Small Angle X-ray Scattering
genomics
mass spectrometry
keratocyte
insulin resistance
Low Endotoxin Recovery
phase transitions
Megalobrama amblycephala
Bordetella holmesii
Erwinia amylovora
structure
time response
fibroblast
genomic
sialic acid
ESI MS
reptile-associated salmonellosis
thema EDItEUR::M Medicine and Nursing
topic_facet R5-920
RC581-607
LAL and LER
cornea
Aeromonas
tear fluid
protein dynamics
O-antigen
Plesiomonas shigelloides
lipopolysaccharide induced TNF? factor
hepcidin
pJNK
rat
Proteus penneri
lipid A
LPS tolerance
iron regulatory protein 1 (IRP1)
MALDI-TOF MS
outer core oligosaccharide
inhibitor design
phagosome
lipopolysaccharide
NMR spectroscopy
Bordetellae
respiratory syncytial virus
dendritic cell
MAT
GT-B
polysorbate
Edwardsiella tarda
LPS
innate immune
d-galactan I
inflammation
aspirin
lipopolysaccharide (LPS)
collagen
heptosyltransferase
rhinovirus
serum resistance
hypothalamic inflammation
NMR
IL-6/JAK2/STAT3 pathway
glycosyltransferase
toll-like receptor
core oligosaccharide
Coxiella burnetii
virenose
oxidative stress
ESI FT-ICR
endotoxin
Salmonella
P65 (nuclear factor-?B)
nitric oxide (NO)
allergic respiratory disorder
bacteria
adhesion molecule
ESI MSn
endotoxaemia
HR-MAS
double-stranded RNA
epithelial cell
mouse
lipopolysaccharide stimulation
complement
inner core oligosaccharide
LPS aggregates
Q fever
O-acetylation
sepsis
chemokine
hygiene hypothesis
anti-conjugate serum
structural determination
Small Angle X-ray Scattering
genomics
mass spectrometry
keratocyte
insulin resistance
Low Endotoxin Recovery
phase transitions
Megalobrama amblycephala
Bordetella holmesii
Erwinia amylovora
structure
time response
fibroblast
genomic
sialic acid
ESI MS
reptile-associated salmonellosis
thema EDItEUR::M Medicine and Nursing
url 44738
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