Manipulation of the host cell by viral auxiliary proteins

Productive HIV infection requires completion of all the steps of the replication cycle, the success of which largely relying on the multiple interactions established by viral proteins with cellular partners. Indeed, cellular and viral fates are intertwined and this interplay may involve rerouting of...

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Main Authors: Nadine Laguette, Monsef Benkirane
Format: Online
Language:English
Published: Frontiers Media SA 2021
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Online Access:17673
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author Nadine Laguette
Monsef Benkirane
author_browse Monsef Benkirane
Nadine Laguette
author_facet Nadine Laguette
Monsef Benkirane
author_sort Nadine Laguette
collection Directory of Open Access Books
description Productive HIV infection requires completion of all the steps of the replication cycle, the success of which largely relying on the multiple interactions established by viral proteins with cellular partners. Indeed, cellular and viral fates are intertwined and this interplay may involve rerouting of cellular factors/pathways to the benefit of the viral life cycle. To gain a foothold into host cells, HIV has to take advantage of available cellular factories and overcome the numerous potential blocks opposed to its replication while ensuring cellular survival. Viral auxiliary proteins are a perfect paradigm to illustrate the complexity of the relationship between HIV and its host. Although these accessory proteins are mostly unnecessary for viral replication in permissive cells in vitro, they play a crucial role in regulating viral spread ex vivo in non-permissive cells and in vivo in hosts. Most accessory proteins are pleiotropic and instrumental in the counteraction of restriction factors and proteins involved in innate immune response. Several proteins of the "intrinsic" immune system that detect the presence of the assailant and initiate a subsequent immune response, as well as restriction factors that are directly devoted to arresting the replication cycle at precise steps have been characterized. Despite the numerous cellular mechanisms dedicated to preventing viral replication, HIV is able to efficiently replicate in humans. Indeed, as a master regulator of cellular machineries and processes, not only has HIV evolved strategies to avoid triggering of pattern recognition receptors, but HIV has also elaborated ways to counteract host restriction factors, thereby overcoming the hurdles that oppose efficient replication. This review collection is dedicated to the manipulation of host cells by HIV-1 and HIV-2, with a particular focus on viral accessory proteins.
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spelling doab-20.500.12854ir-526842024-04-05T17:30:59Z Manipulation of the host cell by viral auxiliary proteins Nadine Laguette Monsef Benkirane QR1-502 Q1-390 HIV intracellular sensors restriction factors intrinsic immune system Viral accessory proteins thema EDItEUR::P Mathematics and Science::PS Biology, life sciences::PSG Microbiology (non-medical) Productive HIV infection requires completion of all the steps of the replication cycle, the success of which largely relying on the multiple interactions established by viral proteins with cellular partners. Indeed, cellular and viral fates are intertwined and this interplay may involve rerouting of cellular factors/pathways to the benefit of the viral life cycle. To gain a foothold into host cells, HIV has to take advantage of available cellular factories and overcome the numerous potential blocks opposed to its replication while ensuring cellular survival. Viral auxiliary proteins are a perfect paradigm to illustrate the complexity of the relationship between HIV and its host. Although these accessory proteins are mostly unnecessary for viral replication in permissive cells in vitro, they play a crucial role in regulating viral spread ex vivo in non-permissive cells and in vivo in hosts. Most accessory proteins are pleiotropic and instrumental in the counteraction of restriction factors and proteins involved in innate immune response. Several proteins of the "intrinsic" immune system that detect the presence of the assailant and initiate a subsequent immune response, as well as restriction factors that are directly devoted to arresting the replication cycle at precise steps have been characterized. Despite the numerous cellular mechanisms dedicated to preventing viral replication, HIV is able to efficiently replicate in humans. Indeed, as a master regulator of cellular machineries and processes, not only has HIV evolved strategies to avoid triggering of pattern recognition receptors, but HIV has also elaborated ways to counteract host restriction factors, thereby overcoming the hurdles that oppose efficient replication. This review collection is dedicated to the manipulation of host cells by HIV-1 and HIV-2, with a particular focus on viral accessory proteins. 2021-02-11T18:41:12Z 2021-02-11T18:41:12Z 2015-11-16 15:44:59 2015 book 17673 16648714 9782889194841 https://directory.doabooks.org/handle/20.500.12854/52684 eng Frontiers Research Topics image/jpeg Attribution 4.0 International http://www.frontiersin.org/books/Manipulation_of_the_host_cell_by_viral_auxiliary_proteins/518#nogo http://journal.frontiersin.org/researchtopic/1634/manipulation-of-the-host-cell-by-viral-auxiliary-proteins Frontiers Media SA 10.3389/978-2-88919-484-1 10.3389/978-2-88919-484-1 bf5ce210-e72e-4860-ba9b-c305640ff3ae 9782889194841 118 open access
spellingShingle QR1-502
Q1-390
HIV
intracellular sensors
restriction factors
intrinsic immune system
Viral accessory proteins
thema EDItEUR::P Mathematics and Science::PS Biology, life sciences::PSG Microbiology (non-medical)
Nadine Laguette
Monsef Benkirane
Manipulation of the host cell by viral auxiliary proteins
title Manipulation of the host cell by viral auxiliary proteins
title_full Manipulation of the host cell by viral auxiliary proteins
title_fullStr Manipulation of the host cell by viral auxiliary proteins
title_full_unstemmed Manipulation of the host cell by viral auxiliary proteins
title_short Manipulation of the host cell by viral auxiliary proteins
title_sort manipulation of the host cell by viral auxiliary proteins
topic QR1-502
Q1-390
HIV
intracellular sensors
restriction factors
intrinsic immune system
Viral accessory proteins
thema EDItEUR::P Mathematics and Science::PS Biology, life sciences::PSG Microbiology (non-medical)
topic_facet QR1-502
Q1-390
HIV
intracellular sensors
restriction factors
intrinsic immune system
Viral accessory proteins
thema EDItEUR::P Mathematics and Science::PS Biology, life sciences::PSG Microbiology (non-medical)
url 17673
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