Mitochondrial Dysfunction in Aging and Diseases of Aging

This collection of review articles authored by international experts pulls together current information about the role of mitochondria in aging and diseases of aging. Mitochondria are vitally important cellular organelles and undergo their own aging process becoming less efficient in aged animals in...

Ամբողջական նկարագրություն

Պահպանված է:
Մատենագիտական մանրամասներ
Հիմնական հեղինակ: Haas, Richard H.
Ձևաչափ: Online
Լեզու:անգլերեն
Հրապարակվել է: MDPI - Multidisciplinary Digital Publishing Institute 2021
Խորագրեր:
Առցանց հասանելիություն:42549
Ցուցիչներ: Ավելացրեք ցուցիչ
Չկան պիտակներ, Եղեք առաջինը, ով նշում է այս գրառումը!
_version_ 1869517059928358912
author Haas, Richard H.
author_browse Haas, Richard H.
author_facet Haas, Richard H.
author_sort Haas, Richard H.
collection Directory of Open Access Books
description This collection of review articles authored by international experts pulls together current information about the role of mitochondria in aging and diseases of aging. Mitochondria are vitally important cellular organelles and undergo their own aging process becoming less efficient in aged animals including humans. These changes have wide-ranging significance contributing to immune dysfunction (autoimmunity and immune deficiency), inflammation, delayed healing, skin and retinal damage, cancer and most of the degenerative diseases of aging. Mitochondrial aging predisposes to drug toxicity in the geriatric population and to many of the features of normal aging. The research detailed in this book summarizes current understanding of the role of mitochondria in the complex molecular changes of aging, moving on to specific diseases of aging. Mitochondrial dysfunction is an important target for development of treatments for aging and disease. The last article details how exercise is a treatment and combats many features of the aging process.
format Online
id doab-20.500.12854ir-53609
institution Directory of Open Access Books
language eng
publishDate 2021
publishDateRange 2021
publishDateSort 2021
publisher MDPI - Multidisciplinary Digital Publishing Institute
publisherStr MDPI - Multidisciplinary Digital Publishing Institute
record_format ojs
spelling doab-20.500.12854ir-536092024-04-05T12:32:02Z Mitochondrial Dysfunction in Aging and Diseases of Aging Haas, Richard H. QH301-705.5 Q1-390 age-related diseases n/a sphingolipids glaucoma ALS neurodegeneration mitochondrial dysfunction adaptive immunity senescence de-emergence innate immunity cell danger response mitochondrial transfer axonal transport cytokines mitochondrial age-related macular degeneration prevention heart failure purinergic signaling autophagy Alzheimer’s disease diabetic retinopathy proteostasis 1 immunosenescence Miro1 ROS metabolism optic nerve polypharmacy eIF2? Parkin coenzyme Q10 neurodegenerative disease DNA damage skin exercise nucleotide metabolism pasteur effect stress response inflammation retina drug-induced mitochondrial toxicity neuroinflammation exosomes reactive oxygen species 25(OH)D cardiomyopathy crabtree effect insulin resistance cardiovascular disease ageing genetic mutations metabokines mitochondria multiple sclerosis aerobic healing cycle SOD1 mitophagy PINK1 type 2 diabetes integrated cell stress response morbidity and mortality ultraviolet photoageing cancer aging 25(OH)2D lysosome NAD+ Parkinson’s disease thema EDItEUR::P Mathematics and Science::PS Biology, life sciences This collection of review articles authored by international experts pulls together current information about the role of mitochondria in aging and diseases of aging. Mitochondria are vitally important cellular organelles and undergo their own aging process becoming less efficient in aged animals including humans. These changes have wide-ranging significance contributing to immune dysfunction (autoimmunity and immune deficiency), inflammation, delayed healing, skin and retinal damage, cancer and most of the degenerative diseases of aging. Mitochondrial aging predisposes to drug toxicity in the geriatric population and to many of the features of normal aging. The research detailed in this book summarizes current understanding of the role of mitochondria in the complex molecular changes of aging, moving on to specific diseases of aging. Mitochondrial dysfunction is an important target for development of treatments for aging and disease. The last article details how exercise is a treatment and combats many features of the aging process. 2021-02-11T19:48:05Z 2021-02-11T19:48:05Z 2019-12-09 11:49:15 2019 book 42549 9783039213283 9783039213276 https://directory.doabooks.org/handle/20.500.12854/53609 eng application/octet-stream Attribution-NonCommercial-NoDerivatives 4.0 International https://mdpi.com/books/pdfview/book/1606 MDPI - Multidisciplinary Digital Publishing Institute 10.3390/books978-3-03921-328-3 10.3390/books978-3-03921-328-3 46cabcaa-dd94-4bfe-87b4-55023c1b36d0 9783039213283 9783039213276 270 open access
spellingShingle QH301-705.5
Q1-390
age-related diseases
n/a
sphingolipids
glaucoma
ALS
neurodegeneration
mitochondrial dysfunction
adaptive immunity
senescence
de-emergence
innate immunity
cell danger response
mitochondrial transfer
axonal transport
cytokines
mitochondrial
age-related macular degeneration
prevention
heart failure
purinergic signaling
autophagy
Alzheimer’s disease
diabetic retinopathy
proteostasis
1
immunosenescence
Miro1
ROS
metabolism
optic nerve
polypharmacy
eIF2?
Parkin
coenzyme Q10
neurodegenerative disease
DNA damage
skin
exercise
nucleotide metabolism
pasteur effect
stress response
inflammation
retina
drug-induced mitochondrial toxicity
neuroinflammation
exosomes
reactive oxygen species
25(OH)D
cardiomyopathy
crabtree effect
insulin resistance
cardiovascular disease
ageing
genetic mutations
metabokines
mitochondria
multiple sclerosis
aerobic
healing cycle
SOD1
mitophagy
PINK1
type 2 diabetes
integrated cell stress response
morbidity and mortality
ultraviolet
photoageing
cancer
aging
25(OH)2D
lysosome
NAD+
Parkinson’s disease
thema EDItEUR::P Mathematics and Science::PS Biology, life sciences
Haas, Richard H.
Mitochondrial Dysfunction in Aging and Diseases of Aging
title Mitochondrial Dysfunction in Aging and Diseases of Aging
title_full Mitochondrial Dysfunction in Aging and Diseases of Aging
title_fullStr Mitochondrial Dysfunction in Aging and Diseases of Aging
title_full_unstemmed Mitochondrial Dysfunction in Aging and Diseases of Aging
title_short Mitochondrial Dysfunction in Aging and Diseases of Aging
title_sort mitochondrial dysfunction in aging and diseases of aging
topic QH301-705.5
Q1-390
age-related diseases
n/a
sphingolipids
glaucoma
ALS
neurodegeneration
mitochondrial dysfunction
adaptive immunity
senescence
de-emergence
innate immunity
cell danger response
mitochondrial transfer
axonal transport
cytokines
mitochondrial
age-related macular degeneration
prevention
heart failure
purinergic signaling
autophagy
Alzheimer’s disease
diabetic retinopathy
proteostasis
1
immunosenescence
Miro1
ROS
metabolism
optic nerve
polypharmacy
eIF2?
Parkin
coenzyme Q10
neurodegenerative disease
DNA damage
skin
exercise
nucleotide metabolism
pasteur effect
stress response
inflammation
retina
drug-induced mitochondrial toxicity
neuroinflammation
exosomes
reactive oxygen species
25(OH)D
cardiomyopathy
crabtree effect
insulin resistance
cardiovascular disease
ageing
genetic mutations
metabokines
mitochondria
multiple sclerosis
aerobic
healing cycle
SOD1
mitophagy
PINK1
type 2 diabetes
integrated cell stress response
morbidity and mortality
ultraviolet
photoageing
cancer
aging
25(OH)2D
lysosome
NAD+
Parkinson’s disease
thema EDItEUR::P Mathematics and Science::PS Biology, life sciences
topic_facet QH301-705.5
Q1-390
age-related diseases
n/a
sphingolipids
glaucoma
ALS
neurodegeneration
mitochondrial dysfunction
adaptive immunity
senescence
de-emergence
innate immunity
cell danger response
mitochondrial transfer
axonal transport
cytokines
mitochondrial
age-related macular degeneration
prevention
heart failure
purinergic signaling
autophagy
Alzheimer’s disease
diabetic retinopathy
proteostasis
1
immunosenescence
Miro1
ROS
metabolism
optic nerve
polypharmacy
eIF2?
Parkin
coenzyme Q10
neurodegenerative disease
DNA damage
skin
exercise
nucleotide metabolism
pasteur effect
stress response
inflammation
retina
drug-induced mitochondrial toxicity
neuroinflammation
exosomes
reactive oxygen species
25(OH)D
cardiomyopathy
crabtree effect
insulin resistance
cardiovascular disease
ageing
genetic mutations
metabokines
mitochondria
multiple sclerosis
aerobic
healing cycle
SOD1
mitophagy
PINK1
type 2 diabetes
integrated cell stress response
morbidity and mortality
ultraviolet
photoageing
cancer
aging
25(OH)2D
lysosome
NAD+
Parkinson’s disease
thema EDItEUR::P Mathematics and Science::PS Biology, life sciences
url 42549
work_keys_str_mv AT haasrichardh mitochondrialdysfunctioninaginganddiseasesofaging