Muscle-Tendon-Innervation Unit: Degeneration and Aging - Pathophysiological and Regeneration Mechanisms

Aging is characterized by progressive deterioration of walking ability. This function loss has multiple causes including central and peripheral nerve dysfunction, loss of muscle mass and strength, as well as joints and bone alterations. Muscle-tendon unit and its innervation has a pivotal role in mo...

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Asıl Yazarlar: Paolo Bonaldo, Cesare Faldini, Luciano Merlini
Materyal Türü: Online
Dil:İngilizce
Baskı/Yayın Bilgisi: Frontiers Media SA 2021
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Online Erişim:22954
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author Paolo Bonaldo
Cesare Faldini
Luciano Merlini
author_browse Cesare Faldini
Luciano Merlini
Paolo Bonaldo
author_facet Paolo Bonaldo
Cesare Faldini
Luciano Merlini
author_sort Paolo Bonaldo
collection Directory of Open Access Books
description Aging is characterized by progressive deterioration of walking ability. This function loss has multiple causes including central and peripheral nerve dysfunction, loss of muscle mass and strength, as well as joints and bone alterations. Muscle-tendon unit and its innervation has a pivotal role in motor function performance that can be disrupted by overuse degeneration and aging. Research has shown that overuse degeneration and aging also share some pathophysiological mechanisms including mitochondrial dysfunction, increased apoptosis, abnormal modulation of autophagy, decline in satellite cells, increased generation of reactive oxygen species, and modification of signalling and stress response pathways. This Research Topic is intended to bring together basic researchers and clinicians working in the area of neuroscience, aging, sarcopenia and orthopaedics in human and in animal models. The aim of this cross-fertilization is to accelerate our understanding of the mechanisms involved in aging and degeneration of the muscle-tendon unit and its innervation and to explore the therapeutic potential of pharmacological and physical therapy interventions.
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language eng
publishDate 2021
publishDateRange 2021
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publisherStr Frontiers Media SA
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spelling doab-20.500.12854ir-540992024-04-05T12:35:56Z Muscle-Tendon-Innervation Unit: Degeneration and Aging - Pathophysiological and Regeneration Mechanisms Paolo Bonaldo Cesare Faldini Luciano Merlini RC321-571 Q1-390 Nanoparticles congenital muscular dystrophy Stem/progenitor cells therapy Aging Parkinson's camptocormia Muscle innervation Animal Models Tendon thema EDItEUR::P Mathematics and Science::PS Biology, life sciences::PSA Life sciences: general issues::PSAN Neurosciences Aging is characterized by progressive deterioration of walking ability. This function loss has multiple causes including central and peripheral nerve dysfunction, loss of muscle mass and strength, as well as joints and bone alterations. Muscle-tendon unit and its innervation has a pivotal role in motor function performance that can be disrupted by overuse degeneration and aging. Research has shown that overuse degeneration and aging also share some pathophysiological mechanisms including mitochondrial dysfunction, increased apoptosis, abnormal modulation of autophagy, decline in satellite cells, increased generation of reactive oxygen species, and modification of signalling and stress response pathways. This Research Topic is intended to bring together basic researchers and clinicians working in the area of neuroscience, aging, sarcopenia and orthopaedics in human and in animal models. The aim of this cross-fertilization is to accelerate our understanding of the mechanisms involved in aging and degeneration of the muscle-tendon unit and its innervation and to explore the therapeutic potential of pharmacological and physical therapy interventions. 2021-02-11T20:21:07Z 2021-02-11T20:21:07Z 2017-07-06 13:27:36 2017 book 22954 16648714 9782889451036 https://directory.doabooks.org/handle/20.500.12854/54099 eng Frontiers Research Topics image/jpeg Attribution 4.0 International http://www.frontiersin.org/books/Muscle-Tendon-Innervation_Unit_Degeneration_and_Aging_-_Pathophysiological_and_Regeneration_Mechani/1130#nogo http://journal.frontiersin.org/researchtopic/3777/muscle-tendon-innervation-unit-degeneration-and-aging---pathophysiological-and-regeneration-mechanis Frontiers Media SA 10.3389/978-2-88945-103-6 10.3389/978-2-88945-103-6 bf5ce210-e72e-4860-ba9b-c305640ff3ae 9782889451036 104 open access
spellingShingle RC321-571
Q1-390
Nanoparticles
congenital muscular dystrophy
Stem/progenitor cells
therapy
Aging
Parkinson's camptocormia
Muscle
innervation
Animal Models
Tendon
thema EDItEUR::P Mathematics and Science::PS Biology, life sciences::PSA Life sciences: general issues::PSAN Neurosciences
Paolo Bonaldo
Cesare Faldini
Luciano Merlini
Muscle-Tendon-Innervation Unit: Degeneration and Aging - Pathophysiological and Regeneration Mechanisms
title Muscle-Tendon-Innervation Unit: Degeneration and Aging - Pathophysiological and Regeneration Mechanisms
title_full Muscle-Tendon-Innervation Unit: Degeneration and Aging - Pathophysiological and Regeneration Mechanisms
title_fullStr Muscle-Tendon-Innervation Unit: Degeneration and Aging - Pathophysiological and Regeneration Mechanisms
title_full_unstemmed Muscle-Tendon-Innervation Unit: Degeneration and Aging - Pathophysiological and Regeneration Mechanisms
title_short Muscle-Tendon-Innervation Unit: Degeneration and Aging - Pathophysiological and Regeneration Mechanisms
title_sort muscle tendon innervation unit degeneration and aging pathophysiological and regeneration mechanisms
topic RC321-571
Q1-390
Nanoparticles
congenital muscular dystrophy
Stem/progenitor cells
therapy
Aging
Parkinson's camptocormia
Muscle
innervation
Animal Models
Tendon
thema EDItEUR::P Mathematics and Science::PS Biology, life sciences::PSA Life sciences: general issues::PSAN Neurosciences
topic_facet RC321-571
Q1-390
Nanoparticles
congenital muscular dystrophy
Stem/progenitor cells
therapy
Aging
Parkinson's camptocormia
Muscle
innervation
Animal Models
Tendon
thema EDItEUR::P Mathematics and Science::PS Biology, life sciences::PSA Life sciences: general issues::PSAN Neurosciences
url 22954
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AT cesarefaldini muscletendoninnervationunitdegenerationandagingpathophysiologicalandregenerationmechanisms
AT lucianomerlini muscletendoninnervationunitdegenerationandagingpathophysiologicalandregenerationmechanisms