Mechanisms in Parkinson's Disease

Parkinson's disease (PD) results primarily from the death of dopaminergic neurons in the substantia nigra. Current PD medications treat symptoms; none halt or retard dopaminergic neuron degeneration. The main obstacle to developing neuroprotective therapies is a limited understanding of the key mole...

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collection Directory of Open Access Books
description Parkinson's disease (PD) results primarily from the death of dopaminergic neurons in the substantia nigra. Current PD medications treat symptoms; none halt or retard dopaminergic neuron degeneration. The main obstacle to developing neuroprotective therapies is a limited understanding of the key molecular mechanisms that provoke neurodegeneration. The discovery of PD genes has led to the hypothesis that misfolding of proteins and dysfunction of the ubiquitin-proteasome pathway are pivotal to PD pathogenesis. Previously implicated culprits in PD neurodegeneration, mitochondrial dysfunction, and oxidative stress may also act in part by causing the accumulation of misfolded proteins, in addition to producing other deleterious events in dopaminergic neurons. Neurotoxin-based models have been important in elucidating the molecular cascade of cell death in dopaminergic neurons. PD models based on the manipulation of PD genes should prove valuable in elucidating important aspects of the disease, such as selective vulnerability of substantia nigra dopaminergic neurons to the degenerative process.
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spelling doab-20.500.12854ir-655322024-04-01T14:15:18Z Mechanisms in Parkinson's Disease Dushanova, Juliana Neurology & clinical neurophysiology thema EDItEUR::M Medicine and Nursing::MK Medical specialties, branches of medicine::MKJ Neurology and clinical neurophysiology Parkinson's disease (PD) results primarily from the death of dopaminergic neurons in the substantia nigra. Current PD medications treat symptoms; none halt or retard dopaminergic neuron degeneration. The main obstacle to developing neuroprotective therapies is a limited understanding of the key molecular mechanisms that provoke neurodegeneration. The discovery of PD genes has led to the hypothesis that misfolding of proteins and dysfunction of the ubiquitin-proteasome pathway are pivotal to PD pathogenesis. Previously implicated culprits in PD neurodegeneration, mitochondrial dysfunction, and oxidative stress may also act in part by causing the accumulation of misfolded proteins, in addition to producing other deleterious events in dopaminergic neurons. Neurotoxin-based models have been important in elucidating the molecular cascade of cell death in dopaminergic neurons. PD models based on the manipulation of PD genes should prove valuable in elucidating important aspects of the disease, such as selective vulnerability of substantia nigra dopaminergic neurons to the degenerative process. 2021-04-20T15:19:39Z 2021-04-20T15:19:39Z 2012 book ONIX_20210420_9789533078762_889 9789533078762 9789535167419 https://directory.doabooks.org/handle/20.500.12854/65532 eng image/jpeg n/a https://www.intechopen.com/books https://mts.intechopen.com/storage/books/1360/authors_book/authors_book.pdf IntechOpen IntechOpen 10.5772/1826 10.5772/1826 78a36484-2c0c-47cb-ad67-2b9f5cd4a8f6 9789533078762 9789535167419 IntechOpen 606 open access
spellingShingle Neurology & clinical neurophysiology
thema EDItEUR::M Medicine and Nursing::MK Medical specialties, branches of medicine::MKJ Neurology and clinical neurophysiology
Mechanisms in Parkinson's Disease
title Mechanisms in Parkinson's Disease
title_full Mechanisms in Parkinson's Disease
title_fullStr Mechanisms in Parkinson's Disease
title_full_unstemmed Mechanisms in Parkinson's Disease
title_short Mechanisms in Parkinson's Disease
title_sort mechanisms in parkinson s disease
topic Neurology & clinical neurophysiology
thema EDItEUR::M Medicine and Nursing::MK Medical specialties, branches of medicine::MKJ Neurology and clinical neurophysiology
topic_facet Neurology & clinical neurophysiology
thema EDItEUR::M Medicine and Nursing::MK Medical specialties, branches of medicine::MKJ Neurology and clinical neurophysiology
url ONIX_20210420_9789533078762_889