Arteriogenesis and Therapeutic Neovascularization

For many years, arteriogenesis, also called collateral formation, has been regarded as being a beneficial process to restore blood flow to distal tissues in occluded arteries. Therefore, it is frequently referred to in relation to therapeutic angiogenesis. Despite the big clinical potential and the...

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Baskı/Yayın Bilgisi: MDPI - Multidisciplinary Digital Publishing Institute 2021
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Online Erişim:ONIX_20210501_9783039365937_1002
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collection Directory of Open Access Books
description For many years, arteriogenesis, also called collateral formation, has been regarded as being a beneficial process to restore blood flow to distal tissues in occluded arteries. Therefore, it is frequently referred to in relation to therapeutic angiogenesis. Despite the big clinical potential and the many promising clinical trials on arteriogenesis and therapeutic angiogenesis, the exact molecular mechanisms involved in the multifactorial processes of arteriogenesis are still not completely understood. A better understanding is needed in order to define successful clinical therapies. In this Special Issue, multiple aspects of arteriogenesis and therapeutic angiogenesis will be addressed, ranging from the role of inflammatory processes and immune cells, to growth factors, microRNAs and environmental factors like hypoxia. Therapeutic angiogenesis will also be discussed in relation to the atherosclerosis and intraplaque angiogenesis in hypoxic lesions, as well as specific forms of arteriogenesis in relation to spinal cord blood supply and aorta surgery. The effects of exercise, a frequently prescribed therapy for PAD patients, on arteriogenesis are also discussed. Overall, the papers in this Special Issue on arteriogenesis and therapeutic angiogenesis provide important new insights in the underlying pathophysiological mechanism of these complex processes and may be helpful to define a successful future intervention directed at therapeutic angiogenesis.
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spelling doab-20.500.12854ir-692562024-03-30T23:22:28Z Arteriogenesis and Therapeutic Neovascularization Deindl, Elisabeth Quax, Paul Waltenberger, Johannes Schmitz-Rixen, Thomas factor VII activating protease HABP2 VEGF matrigel neo-vascularization hind limb ischemia angiogenesis arteriogenesis ERK endothelial cells inflammation macrophages atherosclerosis pericyte rAAV capillary microRNA isomiRs epitranscriptome neovascularization A-to-I editing m6A RNA modifications RNA methylation lower extremity arterial disease peripheral arterial disease blood flow restriction activity-based benefits training effects effect mechanism hyperoxygenation vein graft disease vascular biology spinal cord ischemia paraplegia aortic disease TAAA collateral network paraspinous compartment NO NOTCH innate immunity mast cell GH and eNOS IGF-I oxidative stress and arterial inflammation vascular homeostasis GHAS trial collateral artery growth SMC proliferation potassium channel KV1.3 KCa3.1 FGFR-1 Egr-1 PDFG-R αSM-actin TLR2/6 femoral artery ligation blood flow recovery collateral growth VHL loss of function microRNA-212/132 n/a thema EDItEUR::M Medicine and Nursing For many years, arteriogenesis, also called collateral formation, has been regarded as being a beneficial process to restore blood flow to distal tissues in occluded arteries. Therefore, it is frequently referred to in relation to therapeutic angiogenesis. Despite the big clinical potential and the many promising clinical trials on arteriogenesis and therapeutic angiogenesis, the exact molecular mechanisms involved in the multifactorial processes of arteriogenesis are still not completely understood. A better understanding is needed in order to define successful clinical therapies. In this Special Issue, multiple aspects of arteriogenesis and therapeutic angiogenesis will be addressed, ranging from the role of inflammatory processes and immune cells, to growth factors, microRNAs and environmental factors like hypoxia. Therapeutic angiogenesis will also be discussed in relation to the atherosclerosis and intraplaque angiogenesis in hypoxic lesions, as well as specific forms of arteriogenesis in relation to spinal cord blood supply and aorta surgery. The effects of exercise, a frequently prescribed therapy for PAD patients, on arteriogenesis are also discussed. Overall, the papers in this Special Issue on arteriogenesis and therapeutic angiogenesis provide important new insights in the underlying pathophysiological mechanism of these complex processes and may be helpful to define a successful future intervention directed at therapeutic angiogenesis. 2021-05-01T15:45:06Z 2021-05-01T15:45:06Z 2020 book ONIX_20210501_9783039365937_1002 9783039365937 9783039365944 https://directory.doabooks.org/handle/20.500.12854/69256 eng application/octet-stream Attribution 4.0 International https://mdpi.com/books/pdfview/book/3044 https://mdpi.com/books/pdfview/book/3044 MDPI - Multidisciplinary Digital Publishing Institute 10.3390/books978-3-03936-594-4 10.3390/books978-3-03936-594-4 46cabcaa-dd94-4bfe-87b4-55023c1b36d0 9783039365937 9783039365944 220 Basel, Switzerland open access
spellingShingle factor VII activating protease
HABP2
VEGF
matrigel
neo-vascularization
hind limb ischemia
angiogenesis
arteriogenesis
ERK
endothelial cells
inflammation
macrophages
atherosclerosis
pericyte
rAAV
capillary
microRNA
isomiRs
epitranscriptome
neovascularization
A-to-I editing
m6A
RNA modifications
RNA methylation
lower extremity arterial disease
peripheral arterial disease
blood flow restriction
activity-based benefits
training effects
effect mechanism
hyperoxygenation
vein graft disease
vascular biology
spinal cord ischemia
paraplegia
aortic disease
TAAA
collateral network
paraspinous compartment
NO
NOTCH
innate immunity
mast cell
GH and eNOS
IGF-I
oxidative stress and arterial inflammation
vascular homeostasis
GHAS trial
collateral artery growth
SMC proliferation
potassium channel
KV1.3
KCa3.1
FGFR-1
Egr-1
PDFG-R
αSM-actin
TLR2/6
femoral artery ligation
blood flow recovery
collateral growth
VHL loss of function
microRNA-212/132
n/a
thema EDItEUR::M Medicine and Nursing
Arteriogenesis and Therapeutic Neovascularization
title Arteriogenesis and Therapeutic Neovascularization
title_full Arteriogenesis and Therapeutic Neovascularization
title_fullStr Arteriogenesis and Therapeutic Neovascularization
title_full_unstemmed Arteriogenesis and Therapeutic Neovascularization
title_short Arteriogenesis and Therapeutic Neovascularization
title_sort arteriogenesis and therapeutic neovascularization
topic factor VII activating protease
HABP2
VEGF
matrigel
neo-vascularization
hind limb ischemia
angiogenesis
arteriogenesis
ERK
endothelial cells
inflammation
macrophages
atherosclerosis
pericyte
rAAV
capillary
microRNA
isomiRs
epitranscriptome
neovascularization
A-to-I editing
m6A
RNA modifications
RNA methylation
lower extremity arterial disease
peripheral arterial disease
blood flow restriction
activity-based benefits
training effects
effect mechanism
hyperoxygenation
vein graft disease
vascular biology
spinal cord ischemia
paraplegia
aortic disease
TAAA
collateral network
paraspinous compartment
NO
NOTCH
innate immunity
mast cell
GH and eNOS
IGF-I
oxidative stress and arterial inflammation
vascular homeostasis
GHAS trial
collateral artery growth
SMC proliferation
potassium channel
KV1.3
KCa3.1
FGFR-1
Egr-1
PDFG-R
αSM-actin
TLR2/6
femoral artery ligation
blood flow recovery
collateral growth
VHL loss of function
microRNA-212/132
n/a
thema EDItEUR::M Medicine and Nursing
topic_facet factor VII activating protease
HABP2
VEGF
matrigel
neo-vascularization
hind limb ischemia
angiogenesis
arteriogenesis
ERK
endothelial cells
inflammation
macrophages
atherosclerosis
pericyte
rAAV
capillary
microRNA
isomiRs
epitranscriptome
neovascularization
A-to-I editing
m6A
RNA modifications
RNA methylation
lower extremity arterial disease
peripheral arterial disease
blood flow restriction
activity-based benefits
training effects
effect mechanism
hyperoxygenation
vein graft disease
vascular biology
spinal cord ischemia
paraplegia
aortic disease
TAAA
collateral network
paraspinous compartment
NO
NOTCH
innate immunity
mast cell
GH and eNOS
IGF-I
oxidative stress and arterial inflammation
vascular homeostasis
GHAS trial
collateral artery growth
SMC proliferation
potassium channel
KV1.3
KCa3.1
FGFR-1
Egr-1
PDFG-R
αSM-actin
TLR2/6
femoral artery ligation
blood flow recovery
collateral growth
VHL loss of function
microRNA-212/132
n/a
thema EDItEUR::M Medicine and Nursing
url ONIX_20210501_9783039365937_1002