New Perspective in Atrial Fibrillation

In spite of the large volume of associated research, the pathophysiological mechanisms involved in atrial fibrillation (AF) onset and recurrence remain uncertain. This may explain why the performances of thromboembolic and bleeding prediction scores in AF patients are limited. In the past few years,...

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Опубликовано: MDPI - Multidisciplinary Digital Publishing Institute 2022
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Online-ссылка:ONIX_20220111_9783036518220_454
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collection Directory of Open Access Books
description In spite of the large volume of associated research, the pathophysiological mechanisms involved in atrial fibrillation (AF) onset and recurrence remain uncertain. This may explain why the performances of thromboembolic and bleeding prediction scores in AF patients are limited. In the past few years, the concept of atrial cardiopathy has emerged as a promising lead to connect AF to stroke, heart failure, and inflammatory processes: indeed, all of the mechanisms associated with atrial remodeling and the development of atrial cardiopathy are also likely to promote the development of AF. This recent concept of atrial cardiopathy suggests that the real trigger of stroke may be an abnormal atrial substrate rather than atrial rhythm itself. In this setting, AF could be seen as a symptom of atrial cardiopathy rather than a risk factor of stroke. In the absence of validated clinical markers of atrial cardiopathy, the search for the mechanism of AF remains the cornerstone of cardioembolic stroke prevention for now.The aim of this Special Issue is to gather basic research as well as pathophysiological and epidemiological papers focused on the relationship between atrial substrates and atrial fibrillation onset, recurrence, and outcomes.
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spelling doab-20.500.12854ir-767192024-03-31T13:10:20Z New Perspective in Atrial Fibrillation Guenancia, Charles postoperative atrial fibrillation biomarkers coronary artery bypass grafting miRNA circRNA mtDNA SNPs atrial fibrillation acute myocardial infarction heart rate variability autonomic nervous system bleeding risk age left atrial appendage closure coronary artery disease prognosis anticoagulation antiplatelet therapy hypertension elderly prevention ganglionated plexi ablation dementia early-onset n/a ischemic stroke risk factor new oral anticoagulants mitochondria cardiac remodeling pharmacotherapy electroporation pulsed field ablation cardiac heart arrhythmia NOAC VKA statin outcome mortality paroxysmal atrial fibrillation catheter ablation epicardial adipose tissue persistent atrial fibrillation posterior wall hybrid ablation convergent ablation atrial tachycardia mechanism spatiotemporal dispersion left atrial appendage occlusion BNP atrial remodeling atrial cardiopathy cardiac ablation irreversible electroporation thema EDItEUR::M Medicine and Nursing In spite of the large volume of associated research, the pathophysiological mechanisms involved in atrial fibrillation (AF) onset and recurrence remain uncertain. This may explain why the performances of thromboembolic and bleeding prediction scores in AF patients are limited. In the past few years, the concept of atrial cardiopathy has emerged as a promising lead to connect AF to stroke, heart failure, and inflammatory processes: indeed, all of the mechanisms associated with atrial remodeling and the development of atrial cardiopathy are also likely to promote the development of AF. This recent concept of atrial cardiopathy suggests that the real trigger of stroke may be an abnormal atrial substrate rather than atrial rhythm itself. In this setting, AF could be seen as a symptom of atrial cardiopathy rather than a risk factor of stroke. In the absence of validated clinical markers of atrial cardiopathy, the search for the mechanism of AF remains the cornerstone of cardioembolic stroke prevention for now.The aim of this Special Issue is to gather basic research as well as pathophysiological and epidemiological papers focused on the relationship between atrial substrates and atrial fibrillation onset, recurrence, and outcomes. 2022-01-11T13:40:03Z 2022-01-11T13:40:03Z 2021 book ONIX_20220111_9783036518220_454 9783036518220 9783036518213 https://directory.doabooks.org/handle/20.500.12854/76719 eng image/jpeg Attribution 4.0 International https://mdpi.com/books/pdfview/book/4168 https://mdpi.com/books/pdfview/book/4168 MDPI - Multidisciplinary Digital Publishing Institute 10.3390/books978-3-0365-1821-3 10.3390/books978-3-0365-1821-3 46cabcaa-dd94-4bfe-87b4-55023c1b36d0 9783036518220 9783036518213 224 Basel, Switzerland open access
spellingShingle postoperative atrial fibrillation
biomarkers
coronary artery bypass grafting
miRNA
circRNA
mtDNA
SNPs
atrial fibrillation
acute myocardial infarction
heart rate variability
autonomic nervous system
bleeding risk
age
left atrial appendage closure
coronary artery disease
prognosis
anticoagulation
antiplatelet therapy
hypertension
elderly
prevention
ganglionated plexi
ablation
dementia
early-onset
n/a
ischemic stroke
risk factor
new oral anticoagulants
mitochondria
cardiac remodeling
pharmacotherapy
electroporation
pulsed field ablation
cardiac
heart
arrhythmia
NOAC
VKA
statin
outcome
mortality
paroxysmal atrial fibrillation
catheter ablation
epicardial adipose tissue
persistent atrial fibrillation
posterior wall
hybrid ablation
convergent ablation
atrial tachycardia
mechanism
spatiotemporal dispersion
left atrial appendage occlusion
BNP
atrial remodeling
atrial cardiopathy
cardiac ablation
irreversible electroporation
thema EDItEUR::M Medicine and Nursing
New Perspective in Atrial Fibrillation
title New Perspective in Atrial Fibrillation
title_full New Perspective in Atrial Fibrillation
title_fullStr New Perspective in Atrial Fibrillation
title_full_unstemmed New Perspective in Atrial Fibrillation
title_short New Perspective in Atrial Fibrillation
title_sort new perspective in atrial fibrillation
topic postoperative atrial fibrillation
biomarkers
coronary artery bypass grafting
miRNA
circRNA
mtDNA
SNPs
atrial fibrillation
acute myocardial infarction
heart rate variability
autonomic nervous system
bleeding risk
age
left atrial appendage closure
coronary artery disease
prognosis
anticoagulation
antiplatelet therapy
hypertension
elderly
prevention
ganglionated plexi
ablation
dementia
early-onset
n/a
ischemic stroke
risk factor
new oral anticoagulants
mitochondria
cardiac remodeling
pharmacotherapy
electroporation
pulsed field ablation
cardiac
heart
arrhythmia
NOAC
VKA
statin
outcome
mortality
paroxysmal atrial fibrillation
catheter ablation
epicardial adipose tissue
persistent atrial fibrillation
posterior wall
hybrid ablation
convergent ablation
atrial tachycardia
mechanism
spatiotemporal dispersion
left atrial appendage occlusion
BNP
atrial remodeling
atrial cardiopathy
cardiac ablation
irreversible electroporation
thema EDItEUR::M Medicine and Nursing
topic_facet postoperative atrial fibrillation
biomarkers
coronary artery bypass grafting
miRNA
circRNA
mtDNA
SNPs
atrial fibrillation
acute myocardial infarction
heart rate variability
autonomic nervous system
bleeding risk
age
left atrial appendage closure
coronary artery disease
prognosis
anticoagulation
antiplatelet therapy
hypertension
elderly
prevention
ganglionated plexi
ablation
dementia
early-onset
n/a
ischemic stroke
risk factor
new oral anticoagulants
mitochondria
cardiac remodeling
pharmacotherapy
electroporation
pulsed field ablation
cardiac
heart
arrhythmia
NOAC
VKA
statin
outcome
mortality
paroxysmal atrial fibrillation
catheter ablation
epicardial adipose tissue
persistent atrial fibrillation
posterior wall
hybrid ablation
convergent ablation
atrial tachycardia
mechanism
spatiotemporal dispersion
left atrial appendage occlusion
BNP
atrial remodeling
atrial cardiopathy
cardiac ablation
irreversible electroporation
thema EDItEUR::M Medicine and Nursing
url ONIX_20220111_9783036518220_454