NASH and Systemic Complications

Nonalcoholic fatty liver disease (NAFLD) is known as the hepatic manifestation of the metabolic syndrome, and while most patients develop simple steatosis, up to one-third can develop nonalcoholic steatohepatitis (NASH). NASH is a chronic inflammatory condition of the liver that can further progress...

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collection Directory of Open Access Books
description Nonalcoholic fatty liver disease (NAFLD) is known as the hepatic manifestation of the metabolic syndrome, and while most patients develop simple steatosis, up to one-third can develop nonalcoholic steatohepatitis (NASH). NASH is a chronic inflammatory condition of the liver that can further progress to fibrosis and cirrhosis, which may eventually lead to liver failure and death. While we have increased our mechanistic knowledge regarding the pathogenesis of NASH within the last decade, treatment options are still limited and liver biopsies have remained the gold standard for diagnosis. To achieve major clinical breakthroughs for NASH patients, it is not sufficient to use a single animal model, since each model has specific limitations. Furthermore, we should rely more on alternative models such as organ-on-a-chip, which will enable us to explore unknown aspects of disease pathogenesis much faster and serve as clinically relevant surrogates for murine models. Another important direction for the improvement of patient health is to pay more attention to extrahepatic, organ-specific and systemic effects, which are associated with NASH. The articles in this Special Issue include an up-to-date overview of the rapidly developing technologies, novel targets for intervention and insights in the field in NASH. Additionally, these articles describe the major challenges in the field, strategies to overcome them and suggestions for future directions. To improve patient’s outcome, clinicians, as well as scientists with biomedical, nutrition, physics and mathematics backgrounds, should join forces. Although challenges remain, the future of the field seems promising as these novel technologies and developments are expected to lead to progress in NASH.
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spelling doab-20.500.12854ir-788182024-03-27T16:34:44Z NASH and Systemic Complications Shiri-Sverdlov, Ronit Baumgartner, Sabine NAFLD mouse models multifactorial disease translational value non-alcoholic fatty liver disease steatohepatitis NASH cognitive impairment memory dysfunction Alzheimer’s disease neurodegeneration liver stiffness liver steatosis controlled attenuation parameter transient elastography MRI PDFF MR spectroscopy non-alcoholic steatohepatitis (NASH) tunneling nanotubes (TNTs) primary hepatocytes organelle transfer mesenchymal stromal cells aminopyrine microsomal liver function breath test nonalcoholic fatty liver disease nonalcoholic steatohepatitis extracellular vesicles magnesium (Mg2+) obesity insulin resistance (IR) type 2 diabetes mellitus (T2DM) hypertension cardiovascular diseases (CVD) non-alcoholic steatohepatitis intestinal permeability microbiota probiotics physical exercise fecal microbiota transplantation hepatocellular carcinoma pathogenesis extra-hepatic outcome organ-on-a-chip artificial intelligence liver disease GLP-1 agonist SGLT-2 inhibitor type 2 diabetes innate immune system adaptive immune system metabolic syndrome fatty liver leptin n/a thema EDItEUR::G Reference, Information and Interdisciplinary subjects::GP Research and information: general Nonalcoholic fatty liver disease (NAFLD) is known as the hepatic manifestation of the metabolic syndrome, and while most patients develop simple steatosis, up to one-third can develop nonalcoholic steatohepatitis (NASH). NASH is a chronic inflammatory condition of the liver that can further progress to fibrosis and cirrhosis, which may eventually lead to liver failure and death. While we have increased our mechanistic knowledge regarding the pathogenesis of NASH within the last decade, treatment options are still limited and liver biopsies have remained the gold standard for diagnosis. To achieve major clinical breakthroughs for NASH patients, it is not sufficient to use a single animal model, since each model has specific limitations. Furthermore, we should rely more on alternative models such as organ-on-a-chip, which will enable us to explore unknown aspects of disease pathogenesis much faster and serve as clinically relevant surrogates for murine models. Another important direction for the improvement of patient health is to pay more attention to extrahepatic, organ-specific and systemic effects, which are associated with NASH. The articles in this Special Issue include an up-to-date overview of the rapidly developing technologies, novel targets for intervention and insights in the field in NASH. Additionally, these articles describe the major challenges in the field, strategies to overcome them and suggestions for future directions. To improve patient’s outcome, clinicians, as well as scientists with biomedical, nutrition, physics and mathematics backgrounds, should join forces. Although challenges remain, the future of the field seems promising as these novel technologies and developments are expected to lead to progress in NASH. 2022-02-24T10:37:34Z 2022-02-24T10:37:34Z 2022 book ONIX_20220224_9783036529790_116 9783036529790 9783036529783 https://directory.doabooks.org/handle/20.500.12854/78818 eng image/jpeg Attribution 4.0 International https://mdpi.com/books/pdfview/book/4913 https://mdpi.com/books/pdfview/book/4913 MDPI - Multidisciplinary Digital Publishing Institute 10.3390/books978-3-0365-2979-0 10.3390/books978-3-0365-2979-0 46cabcaa-dd94-4bfe-87b4-55023c1b36d0 9783036529790 9783036529783 232 Basel open access
spellingShingle NAFLD
mouse models
multifactorial disease
translational value
non-alcoholic fatty liver disease
steatohepatitis
NASH
cognitive impairment
memory dysfunction
Alzheimer’s disease
neurodegeneration
liver stiffness
liver steatosis
controlled attenuation parameter
transient elastography
MRI PDFF
MR spectroscopy
non-alcoholic steatohepatitis (NASH)
tunneling nanotubes (TNTs)
primary hepatocytes
organelle transfer
mesenchymal stromal cells
aminopyrine
microsomal
liver function
breath test
nonalcoholic fatty liver disease
nonalcoholic steatohepatitis
extracellular vesicles
magnesium (Mg2+)
obesity
insulin resistance (IR)
type 2 diabetes mellitus (T2DM)
hypertension
cardiovascular diseases (CVD)
non-alcoholic steatohepatitis
intestinal permeability
microbiota
probiotics
physical exercise
fecal microbiota transplantation
hepatocellular carcinoma
pathogenesis
extra-hepatic outcome
organ-on-a-chip
artificial intelligence
liver disease
GLP-1 agonist
SGLT-2 inhibitor
type 2 diabetes
innate immune system
adaptive immune system
metabolic syndrome
fatty liver
leptin
n/a
thema EDItEUR::G Reference, Information and Interdisciplinary subjects::GP Research and information: general
NASH and Systemic Complications
title NASH and Systemic Complications
title_full NASH and Systemic Complications
title_fullStr NASH and Systemic Complications
title_full_unstemmed NASH and Systemic Complications
title_short NASH and Systemic Complications
title_sort nash and systemic complications
topic NAFLD
mouse models
multifactorial disease
translational value
non-alcoholic fatty liver disease
steatohepatitis
NASH
cognitive impairment
memory dysfunction
Alzheimer’s disease
neurodegeneration
liver stiffness
liver steatosis
controlled attenuation parameter
transient elastography
MRI PDFF
MR spectroscopy
non-alcoholic steatohepatitis (NASH)
tunneling nanotubes (TNTs)
primary hepatocytes
organelle transfer
mesenchymal stromal cells
aminopyrine
microsomal
liver function
breath test
nonalcoholic fatty liver disease
nonalcoholic steatohepatitis
extracellular vesicles
magnesium (Mg2+)
obesity
insulin resistance (IR)
type 2 diabetes mellitus (T2DM)
hypertension
cardiovascular diseases (CVD)
non-alcoholic steatohepatitis
intestinal permeability
microbiota
probiotics
physical exercise
fecal microbiota transplantation
hepatocellular carcinoma
pathogenesis
extra-hepatic outcome
organ-on-a-chip
artificial intelligence
liver disease
GLP-1 agonist
SGLT-2 inhibitor
type 2 diabetes
innate immune system
adaptive immune system
metabolic syndrome
fatty liver
leptin
n/a
thema EDItEUR::G Reference, Information and Interdisciplinary subjects::GP Research and information: general
topic_facet NAFLD
mouse models
multifactorial disease
translational value
non-alcoholic fatty liver disease
steatohepatitis
NASH
cognitive impairment
memory dysfunction
Alzheimer’s disease
neurodegeneration
liver stiffness
liver steatosis
controlled attenuation parameter
transient elastography
MRI PDFF
MR spectroscopy
non-alcoholic steatohepatitis (NASH)
tunneling nanotubes (TNTs)
primary hepatocytes
organelle transfer
mesenchymal stromal cells
aminopyrine
microsomal
liver function
breath test
nonalcoholic fatty liver disease
nonalcoholic steatohepatitis
extracellular vesicles
magnesium (Mg2+)
obesity
insulin resistance (IR)
type 2 diabetes mellitus (T2DM)
hypertension
cardiovascular diseases (CVD)
non-alcoholic steatohepatitis
intestinal permeability
microbiota
probiotics
physical exercise
fecal microbiota transplantation
hepatocellular carcinoma
pathogenesis
extra-hepatic outcome
organ-on-a-chip
artificial intelligence
liver disease
GLP-1 agonist
SGLT-2 inhibitor
type 2 diabetes
innate immune system
adaptive immune system
metabolic syndrome
fatty liver
leptin
n/a
thema EDItEUR::G Reference, Information and Interdisciplinary subjects::GP Research and information: general
url ONIX_20220224_9783036529790_116