Mutagenesis and Mitochondrial-Associated Pathologies

Reactive oxygen species (ROS) and DNA double-strand breaks can result from mitochondrial defects and external sources, such as ionizing radiation. If not repaired properly, pathogenic mutations are generated. Human diseases resulting from inherited mitochondrial defects manifest in organs that physi...

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Format: Online
Language:English
Published: IntechOpen 2022
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Online Access:ONIX_20220727_9781803551722_210
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collection Directory of Open Access Books
description Reactive oxygen species (ROS) and DNA double-strand breaks can result from mitochondrial defects and external sources, such as ionizing radiation. If not repaired properly, pathogenic mutations are generated. Human diseases resulting from inherited mitochondrial defects manifest in organs that physiologically require a high level of ATP synthesis. These diseases are clinically challenging, but new experimental clinical therapies include gene editing and mitochondrial transplants. Pathogenic ROS-associated cellular damage includes DNA double-strand breaks, and mouse models are now available to study multiple repair pathways. This book discusses the clinical manifestations of mitochondrial diseases in both the eye and the kidney, and presents new insights into double-strand break repair pathways and developmental phenotypes of g-ray-associated ontogenic mutations of Drosophila melanogaste.
format Online
id doab-20.500.12854ir-90314
institution Directory of Open Access Books
language eng
publishDate 2022
publishDateRange 2022
publishDateSort 2022
publisher IntechOpen
publisherStr IntechOpen
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spelling doab-20.500.12854ir-903142024-03-31T22:45:27Z Mutagenesis and Mitochondrial-Associated Pathologies Fasullo, Michael Catala, Angel Medical genetics thema EDItEUR::M Medicine and Nursing::MF Pre-clinical medicine: basic sciences::MFN Medical genetics Reactive oxygen species (ROS) and DNA double-strand breaks can result from mitochondrial defects and external sources, such as ionizing radiation. If not repaired properly, pathogenic mutations are generated. Human diseases resulting from inherited mitochondrial defects manifest in organs that physiologically require a high level of ATP synthesis. These diseases are clinically challenging, but new experimental clinical therapies include gene editing and mitochondrial transplants. Pathogenic ROS-associated cellular damage includes DNA double-strand breaks, and mouse models are now available to study multiple repair pathways. This book discusses the clinical manifestations of mitochondrial diseases in both the eye and the kidney, and presents new insights into double-strand break repair pathways and developmental phenotypes of g-ray-associated ontogenic mutations of Drosophila melanogaste. 2022-07-27T08:19:22Z 2022-07-27T08:19:22Z 2022 book ONIX_20220727_9781803551722_210 9781803551722 9781803551715 9781803551739 https://directory.doabooks.org/handle/20.500.12854/90314 eng image/jpeg n/a https://www.intechopen.com/books/11348 https://mts.intechopen.com/storage/books/11348/authors_book/authors_book.pdf IntechOpen IntechOpen 10.5772/intechopen.98146 10.5772/intechopen.98146 78a36484-2c0c-47cb-ad67-2b9f5cd4a8f6 9781803551722 9781803551715 9781803551739 IntechOpen 116 open access
spellingShingle Medical genetics
thema EDItEUR::M Medicine and Nursing::MF Pre-clinical medicine: basic sciences::MFN Medical genetics
Mutagenesis and Mitochondrial-Associated Pathologies
title Mutagenesis and Mitochondrial-Associated Pathologies
title_full Mutagenesis and Mitochondrial-Associated Pathologies
title_fullStr Mutagenesis and Mitochondrial-Associated Pathologies
title_full_unstemmed Mutagenesis and Mitochondrial-Associated Pathologies
title_short Mutagenesis and Mitochondrial-Associated Pathologies
title_sort mutagenesis and mitochondrial associated pathologies
topic Medical genetics
thema EDItEUR::M Medicine and Nursing::MF Pre-clinical medicine: basic sciences::MFN Medical genetics
topic_facet Medical genetics
thema EDItEUR::M Medicine and Nursing::MF Pre-clinical medicine: basic sciences::MFN Medical genetics
url ONIX_20220727_9781803551722_210